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Elevated cTn in secure CAD may very well be attributed to Improved proteolytic degradation of myofibrillar troponin. Minimized Ca2+ extrusion through the Na/Ca exchanger, with concomitant decreased Ca2+ uptake through the sarcoplasmic reticulum, may create regional elevations from the focus of intracellular Ca2+ in the course of ischaemia.79 With r